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Joined 1 year ago
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Cake day: June 8th, 2023

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  • what they hate is there being cultural aspects not under their control

    china claims they were interning uighurs to prevent extremist terrorism and separatism, not control culture. xinjiang shares a border with afghanistan. the claim is that uighurs were going off to fight in syria and other regions, then coming back to start shit at home. china claims the mass detainment of uighurs was to provide language and vocational training to counter the sway of jihadists returning from the middle east.







  • The issue with this is beehaw is large enough that them defederating from other instances is potentially a serious threat to those instances. Social networks are inherently monopolistic because people follow the crowd, and federation is meant to counteract that tendency toward userbase consolidation. Moves like this could be interpreted as an attempt to become the dominant instance, defeating the purpose of the fediverse.


  • iie@lemmy.mltoPolitics@lemmy.ml"Allow"
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    1 year ago

    The move to extend the exemptions, rather than winding them down, amounts to a recognition by U.S. authorities that efforts to isolate China from high-tech goods are more difficult than anticipated in a highly integrated global industry, according to industry executives.





  • This is really interesting. As the study notes, this is not a general property of viral infections or even viral respiratory infections, and COVID is actually kinda distinctive in this regard:

    Various immune challenges induce diverse immunological responses that differentially involve particular subsets of immune cells and induce disease-specific cytokine and chemokine profiles systemically and in the CNS. The differences in neuroinflammatory profiles observed here in mouse models of SARS-CoV-2 and H1N1 influenza respiratory infection underscore this principle. SARS-CoV-2 infection induces a broad inflammatory response—well beyond the typical type-1 immune response seen with other respiratory viral infections (Lucas et al., 2020). Concordantly, we find here that even mild respiratory COVID can induce prominent elevation of multiple cytokines and chemokines together with lasting reactivity of white matter microglia in subcortical and hippocampal regions. By comparison, H1N1 influenza elicited a partially overlapping but also distinct CSF cytokine/chemokine profile and more restricted pattern of persistent cellular changes.